References

37 (9), 997-998

Starr, L.B., Robinson, R.G. and Price, T.R. (1983), Reliability, Validity, and Clinical Utility of the Social Functioning Exam in the Assessment of Stroke Patients. Experimental Aging Research, 9 (2), 101-106

Wolf, S.L., Lecraw, D.E., Barton, L.A. and Jann, B.B. (1989), Forced Use of Hemiplegic Upper Extremities to Reverse the Effect of Learned Nonuse Among Chronic Stroke and Head-Injured Patients. Experimental Neurology, 104 (2), 125-132

[Anon]. (1983), Pill Cuts Arthritis, Ovarian-Cancer Risk - Doesnt Cause Gallbladder-Disease - Raises Stroke Risk Slightly. Family Planning Perspectives, 15 (1), 36-37

Umegaki, K., Ikeda, M., Nakamura, K. and Tomita, T. (1986), Defects of Thrombin-Induced Protein-Phosphorylation in Platelets from Stroke-Prone Spontaneously Hypertensive Rats. Febs Letters, 196 (1), 139-144

Overgaard, K., Lundbaek, J.A. and Boysen, G. (1990), Thrombolytic Therapy in Stroke - A Rat Model. Fibrinolysis, 4 13-14

Keywords: FIBRINOLYSIS/SCOTLAND

Hirschberg, M. (1990), Dog-Model for Thrombolysis in Acute Ischemic Stroke. Fibrinolysis, 4 9-11

Keywords: FIBRINOLYSIS/SCOTLAND

Abel, D.C., Lowell, W.R., Graham, J.B. and Shabetai, R. (1987), Elasmobranch Pericardial Function .2. the Influence of Pericardial Pressure on Cardiac Stroke Volume in Horn Sharks and Blue Sharks. Fish Physiology and Biochemistry, 4 (1), 5-14

Palets, L.D. (1983), The Cardiac Pump Function, Power and Stroke Work in Intact Animals Under Prolonged Adrenaline Infusion. Fiziologicheskii Zhurnal, 29 (3), 272-279

Zscheile, K. (1984), Suhl Meat Combine Stroke the Balance. Fleisch, 38 (6), 114

Shiota, C., Sasagawa, S., Hara, K., Ikoma, Y. and Oshino, N. (1982), Central Dopaminergic Function in Stroke-Prone Spontaneously Hypertensive Rats (Shrsp) .1. Alteration of Locomotor-Activity and Swimming Ability. Folia Pharmacologica Japonica, 80 (5), 375-384

Hara, K., Ikoma, Y., Nakao, H., Ezumi, K., Oshino, N., Shiota, C. and Sasagawa, S. (1982), Central Dopaminergic Function in Stroke-Prone Spontaneously Hypertensive Rats (Shrsp) .2. Effects of Chronic Treatment with Lisuride on the Impaired Swimming Ability. Folia Pharmacologica Japonica, 80 (5), 385-394

Suno, M., Shibota, M. and Nagaoka, A. (1984), Effect of Cv-2619 (Idebenone) on the Half-Life and Hemolysis of Red-Blood-Cells in Stroke-Prone Spontaneously Hypertensive Rats (Shrsp). Folia Pharmacologica Japonica, 84 (4), 407-410

Bando, K., Hatanaka, Y., Ohbayashi, S. and Irino, O. (1988), A Quantitative Measurement of the Cerebral Infarct Focus Induced by Arachidonate Infusion and the Relationship Between Measured Values and Stroke Signs. Folia Pharmacologica Japonica, 92 (3), 193-200

Yamashita, K., Shigematsu, K., Maeda, T., Ishida, Y., Niwa, M. and Ozaki, M. (1989), Antihypertensive Effect of Bopindolol on Stroke-Prone Spontaneously Hypertensive Rats (Shrsp). Folia Pharmacologica Japonica, 93 (1), 7-15

Uchida, S., Itoh, M., Fujimoto, M., Yamashita, K., Niwa, M., Sekine, I. and Ozaki, M. (1990), Antihypertensive Effects of Betaxolol, A Cardioselective Beta- Adrenoceptor Antagonist, in Stroke-Prone Spontaneously Hypertensive Rats (Shrsp). Folia Pharmacologica Japonica, 96 (2), 49-63

Keywords: JAPAN

Braun, M. (1981), Closed Drainage of Urinary-Bladder on Rehabilitation of the Infected Contracted Bladder After Apoplectic Stroke. Fortschritte der Medizin, 99 (26), 1039-&

Wassmann, H. (1982), Treatment of Completed Stroke with Hyperbaric Oxygenation and Extra-Intracranial Arterial Bypass Operation. Fortschritte der Medizin, 100 (7), 285-288

Durner, A. and Vonalbert, H.H. (1985), The Importance of the Time Factor for Cct-Findings in Completely Reversible Stroke. Fortschritte der Medizin, 103 (29), 721-724

Wessely, P., Zeiler, K., Bianco, P.D. and Mamoli, B. (1981), Relations Between Clinical and Angiographical Results in Cases of Completed Stroke. Fortschritte der Neurologie Psychiatrie, 49 (5), 179-186

Hornig, C.R., Buttner, T. and Dorndorf, W. (1987), Lethality of Ischemic Stroke. Fortschritte der Neurologie Psychiatrie, 55 (11), 347-353

Uttal, B. (1986), How to Cope When the Ceo Has A Stroke. Fortune, 113 (5), 42-44

Fierman, J. (1987), Reed,John Bold Stroke. Fortune, 115 (13), 26-32

Ishii, Y. (1988), Analysis of Kanji Structures and A Method for Recognizing Online Handwritten Kanji Characters by Means of Stroke Representative Points. Fujitsu Scientific & Technical Journal, 24 (3), 177-202

Evrard, S. (1990), The Decline of Stroke. Gazette Medicale, 97 (26), 59-64

Kuhl, H. (1990), Ovulation Preventatives - the Significance of the Estrogen Dose. Geburtshilfe und Frauenheilkunde, 50 (12), 910-922.

Abstract: The large prospective studies on adverse effects of oral contraceptives have unanimously revealed an increased risk of thromboembolic diseases, which seem to be associated with the dose of ethinylestradiol (EE). According to the recommendations of several medical committees, the dose of EE has, therefore, been more and more reduced; in some countries there are now ovulation inhibitors containing 20-mu-g EE. Since serious reactions, which have a relatively low incidence, are highly underreported (less than 10 %), it is difficult to prove dose-dependent differences in the rates of cardiovascular diseases. There is, however, virtually no doubt that not only the incidence of thromboembolic diseases and stroke, but also that of benign liver tumours and gall bladder diseases is increased in relation to the EE-dose. A series of metabolic serum parameters, e.g. serum binding proteins, coagulation and fibrinolysis factors, angiotensinogen, is changed by EE in a dose-dependent manner which is, however, limited when the effects are receptor-mediated. Higher doses of EE have been shown to facilitate fibrin deposits on vascular subendothelium. The pharmacological effects of EE are to a large extent dependent on the dose, e.g. the irreversible reactions of EE and other ethinylated steroids with hepatic enzymes which are involved in the metabolism of steroids, drugs and toxic compounds. After long-term treatment with combinations containing 50-mu-g EE, in half of the women, abnormal liver function tests with pathological morphological alterations have been found. As combinations with low EE doses and a sufficiently effective progestogen component do not differ from higher dosed oral contraceptives in their contractives safety and cycle control, there are no indications for pills containing 50-mu-g EE, except the normophasic sequential preparations for women with sustained irregular bleedings when taking low dose combinations

Keywords: 17 ALPHA-ETHYNYLESTRADIOL/BLOOD-PRESSURE/CARBOHYDRATE-METABOLISM/ETHINYL ESTRADIOL/ETHYNYL ESTROGENS/FUNCTIONAL OVARIAN CYSTS/HUMAN-LIVER-MICROSOMES/ORAL-CONTRACEPTIVE USERS/POST-MENOPAUSAL WOMEN/PROGESTATIONAL AGENTS

Morris, P.L.P. and Raphael, B. (1987), Depressive Disorder Associated with Physical Illness - the Impact of Stroke. General Hospital Psychiatry, 9 (5), 324-330

Kawashima, K., Hayakawa, T., Miwa, Y., Oohata, H., Suzuki, T., Fujimoto, K., Ogino, T. and Chen, Z.X. (1990), Structure and Hypotensive Activity Relationships of Tetrandrine Derivatives in Stroke-Prone Spontaneously Hypertensive Rats. General Pharmacology, 21 (3), 343-347

Idone, V.P., Orville, R.E., Mach, D.M. and Rust, W.D. (1987), The Propagation Speed of A Positive Lightning Return Stroke. Geophysical Research Letters, 14 (11), 1150-1153

Cooray, V. (1989), Derivation of Return Stroke Parameters from the Electric and Magnetic-Field Derivatives. Geophysical Research Letters, 16 (1), 61-64

Rhodes, C. and Krehbiel, P.R. (1989), Interferometric Observations of A Single Stroke Cloud-To-Ground Flash. Geophysical Research Letters, 16 (10), 1169-1172

Pezzella, G., Irace, C., Viola, R., Cicerano, U., Dechiara, S. and Postiglione, A. (1988), Atrial-Fibrillation And/Or Elevated Hematocrit Increase Short- Term Mortality in Patients with Ischemic Stroke. Geriatric Cardiovascular Medicine, 1 (4), 267-269

Carlow, T.J. and Bicknell, J.M. (1981), Stroke and the Eye - Diagnostic Clues and Treatment Approaches. Geriatrics, 36 (12), 28-&

Boshell, B.R. (1981), Diabetes and Stroke - Coping with the Consequences. Geriatrics, 36 (10), 51-&

Mckinney, A.S. (1981), Appropriate Investigation of Stroke and Dementia. Geriatrics, 36 (6), 41-&

Greer, M. (1982), Strategies for Reducing Stroke Frequency. Geriatrics, 37 (9), 36-42

Kotler, M.N., Mintz, G.S. and Segal, B.L. (1983), Two-Dimensional Echocardiography for Stroke Patients. Geriatrics, 38 (12), 57-&

Steinberg, F.U. (1986), Rehabilitating the Older Stroke Patient - Whats Possible. Geriatrics, 41 (3), 85-&

Wessler, S. (1987), Anticoagulants in the Prevention of Embolic Stroke .1. Geriatrics, 42 (2), 29-&

Wessler, S. (1987), Prophylactic Anticoagulants in Managing Embolic Stroke. Geriatrics, 42 (3), 83-86

Dobkin, B.H. (1988), Management of Geriatric Tia and Stroke. Geriatrics, 43 (11), 27-&

Grotta, J.C. (1988), Post-Stroke Management Concerns and Outcomes. Geriatrics, 43 (7), 40-&

Robbins, A.S. (1989), Hypothermia and Heat-Stroke - Protecting the Elderly Patient. Geriatrics, 44 (1), 73-&

Aronow, W.S. (1990), Risk-Factors for Geriatric Stroke - Identification and Follow- Up. Geriatrics, 45 (9), 37-&

Baum, H.M. and Manton, K.G. (1987), National Trends in Stroke-Related Mortality - A Comparison of Multiple Cause Mortality Data with Survey and Other Health Data. Gerontologist, 27 (3), 293-300

Norris, V.K., Stephens, M.A.P. and Kinney, J.M. (1990), The Impact of Family Interactions on Recovery from Stroke - Help Or Hindrance. Gerontologist, 30 (4), 535-542

Keywords: STATE

Lin, W.L. and Essner, E. (1988), Ultrastructural and Permeability Characteristics of Retinal- Vessels in Stroke-Prone Spontaneously Hypertensive Rats. Graefes Archive for Clinical and Experimental Ophthalmology, 226 (6), 559-566

Wintzen, A.R. (1985), The Management of Stroke - Fears and Facts. Haemostasis, 15 (4), 278-282

Prins, M.H., Gelsema, R., Sing, A.K., Vanheerde, L.R. and Denottolander, G.J.H. (1989), Prophylaxis of Deep Venous Thrombosis with A Low-Molecular- Weight Heparin (Kabi-2165/Fragmin) in Stroke Patients. Haemostasis, 19 (5), 245-250

Tavazzi, L. (1990), Heparin in Acute Myocardial-Infarction. Haemostasis, 20 122-128.

Abstract: Thrombolytic therapy has become an established procedure in patients with acute myocardial infarction (AMI). However, the aftercare of such patients is still uncertain. A meta-analysis of twenty trials of intravenous or subcutaneous heparin in AMI performed during the prethrombolysis period indicated a significant reduction in mortality, reinfarction, and stroke in treated patients. More recently, a study (SCATI) aimed at investigating the clinical effects of subcutaneous heparin (12,500 U two times daily) in the setting of thrombolytic therapeutical strategy in AMI, showed a lower in-hospital mortality, a trend towards lesser transient ischemic episodes in patients given streptokinase, and no difference in recurrent infarction rate. Ventricular thrombi were markedly reduced by heparin. Anti-platelet drugs were not permitted in the SCATI. In both GISSI 2 (Gruppo Italiano per lo Studio della Streptochinasi nell'Infarto Miocardico) and International tissue plasminogen activator/streptokinase trials, aspirin was a recommended treatment, and subcutaneous heparin was randomized. No difference in mortality was noted in patients given heparin. In GISSI 2 recurrent ischemic episodes were also similar in treated and control groups, whereas embolic events were reduced by heparin. Major bleedings were rare in all trials. In conclusion, subcutaneous heparin is beneficial in AMI; however, the association with aspirin does not add consistent benefits. Heparin is effective in preventing thrombus formation and embolic complications - such effects are not shared by aspirin

Keywords: ANGIOPLASTY/CARE/DEEP- VEIN THROMBOSIS/EARLY THROMBOLYSIS/HEPARIN/INTRAVENOUS HEPARIN/LEFT-VENTRICULAR THROMBUS/MORTALITY/MYOCARDIAL INFARCTION/RANDOMIZED TRIAL/SUBCUTANEOUS HEPARIN/SURVIVAL/SWITZERLAND/THROMBOLYSIS/TISSUE PLASMINOGEN-ACTIVATOR

Lotto, A., Colombo, A., Talarico, G., Fratianni, G. and Lettino, M. (1990), Heparin and Secondary Prevention of Acute Myocardial-Infarction. Haemostasis, 20 132-141.

Abstract: Patients surviving acute myocardial infarction (AMI) may experience several clinical events (reinfarction, congestive heart failure, sudden death) still responsible for high mortality rates. AMI early complicated by residual angina, left ventricular dysfunction, or malignant arrhythmias has a worse prognosis. Secondary prevention of myocardial infarction and death has been the end point of many clinical trials in the past two decades. It is well known that beta blockers prevent sudden death if administered chronically after AMI. Meta- analysis of controlled randomized trials demonstrated a significant reduction in reinfarction and vascular death with long-term antiplatelet treatment. Oral anticoagulants prevent fatal and non-fatal reinfarction and show a trend towards lower mortality rates, though treated patients have a higher incidence of haemorrhagic events, particularly stroke. Early administration of heparin gave contradictory results on short- term prevention of myocardial infarction and death after AMI. Data on long-term heparin therapy point out a significant reduction in recurrent AMI and a trend towards a decrease in general mortality

Keywords: ASPIRIN/BLOCKADE/CONTROLLED TRIAL/FIBRINOPEPTIDE-A/HEPARIN/MORBIDITY/MORTALITY/MULTICENTER/MYOCARDIAL INFARCTION/PROGNOSIS/REINFARCTION/SECONDARY PREVENTION/SWITZERLAND/THERAPY

Serneri, G.G.N., Amaducci, L., Inzitari, D. and Gensini, G.F. (1990), Low-Dose Heparin in the Prevention of Ischemic Stroke - Presentation of the Experimental Protocol of the Low-Dose Heparin Stroke Prevention Study. Haemostasis, 20 142-144

Keywords: ETHICAL COMMITTEE/EXPERIMENTAL PROTOCOL/INFARCTION/LOW-DOSE HEPARIN STROKE PREVENTION/SWITZERLAND/TICLOPIDINE

Prentice, C.R.M. (1990), Pathogenesis of Thrombosis. Haemostasis, 20 50-59.

Abstract: The pathogenesis of thrombosis involves consideration of two processes: atherosclerosis and thrombosis. Atherosclerosis is a prolonged process in which interaction of lipids and the haemostatic system produces focal lesions at sites of turbulence in arteries. These lesions may rupture, precipitating the complication of thrombosis and leading to complete occlusion of the vessel, causing myocardial infarction, stroke, or acute ischaemia. In veins the atherosclerotic process does not occur, and thrombosis is precipitated by immobility often associated with accidental or surgical trauma. The changes in platelets, coagulation, fibrinolytic systems leading to atherosclerosis and thrombi are reviewed. The role of platelets and coagulation changes in these processes are emphasized by the beneficial effects of therapy, both antiplatelet and anticoagulant therapy. Last, the concept of hypercoagulability leading to accelerated arterial disease and the clinical value of laboratory tests used in its detection are considered

Keywords: ATHEROSCLEROSIS/BLOOD-VISCOSITY/COAGULATION/CORONARY-ARTERY DISEASE/ENDOTHELIAL CELL/FACTOR- VII/FIBRINOGEN/FIBRINOLYSIS/GROWTH-FACTOR/HEMOSTATIC FUNCTION/HYPERCOAGULABILITY/MECHANISM/MYOCARDIAL-INFARCTION/PATHOGENESIS/RISK FACTOR/SWITZERLAND/THROMBOSIS/TISSUE FACTOR

Swanson, A.B. and Swanson, G.D. (1989), Evaluation and Treatment of the Upper Extremity in the Stroke Patient. Hand Clinics, 5 (1), 75-96

Levy, R.L. (1981), Stroke and Orgasmic Cephalgia. Headache, 21 (1), 12-13

Spaccavento, L.J. and Solomon, G.D. (1984), Migraine As An Etiology of Stroke in Young-Adults. Headache, 24 (1), 19-22

Bardwell, A. and Trott, J.A. (1987), Stroke in Migraine As A Consequence of Propranolol. Headache, 27 (7), 381-383

Bernsen, H.J.J.A., Vandevlasakker, C., Verhagen, W.I.M. and Prick, M.J.J. (1990), Basilar Artery Migraine Stroke. Headache, 30 (3), 142-144

Spierings, E.L.H. (1990), Angiographic Changes Suggestive of Vasospasm in Migraine Complicated by Stroke. Headache, 30 (11), 727-728.

Abstract: A 30-year-old woman with a history of common migraine developed a permanent left homonymous hemianopia during a typical headache. CT scan demonstrated a right posterior cerebral infarction and angiography showed irregular narrowing of the ipsilateral posterior cerebral artery, suggestive of vasospasm. In the case no risk factors for atherosclerotic stroke were present except for smoking, and no other causes of stroke could be found

Keywords: COMPLICATED MIGRAINE/CT/HEADACHE/STROKE/VASOSPASM/WOMEN

Watson, B. (1983), Different Stroke Therapy. Health, 15 (4), 19

[Anon]. (1984), Lucky Stroke. Health, 16 (5), 64-67

Combe, J., Poinsard, P., Besancenot, J., Cattin, F., Bonneville, J.F. and Camelot, G. (1990), Intraluminal Thrombi of the Cervical Carotid-Artery. Helvetica Chirurgica Acta, 57 (2), 219-221.

Abstract: Intraluminal clot of the internal cervical carotid artery is commonly thought to require emergency surgery: 7 intraluminal clots specially threatening (6 of whom had a long defect - 15 mm and more) are demonstrated by intraarterial digital angiography - 4 patients experienced mild stroke, 3 major stroke. 3 of whom had previous recurrent T.I.A. (3 transient blindness, 1 hemispheric TIA). Carotid angiography identified 3 severe atherosclerotic stenosis, 3 ulcerated plaques and 1 dissection. One patient with coma carus died quickly. Anticoagulation therapy (6 cases) was made, 4 weeks along, without neurologic complications. Follow-up angiograms showed total resolution (4 cases), partial lysis (1 case) and mild extension (1 case). Delayed endarterectomy was made only for severe carotid atheroclerosis (5 cases). In our experience, intraluminal clot of the carotid artery may not be a surgical emergency but require anticoagulant therapy and delayed surgical if major underlying lesions

Keywords: SWITZERLAND/TIA

Ernst, E., Matrai, A., Schonhaber, H., Paulsen, H.F. and Magyarosy, I. (1986), Hemorheological Changes During Stroke Rehabilitation. Herz Kreislauf, 18 (1), 30-34

Buschhaus, J., Trampisch, H.J., Minning, E., Scharfbornhofen, E. and Blumchen, G. (1986), Reproducibility of Cardiac-Output and Stroke Volume Measurements Using Thermodilution. Herz Kreislauf, 18 (12), 615-623

Stix, P. and Egger, J. (1987), The Pathway to the Hospital - Differences Between Patients with Stroke and Cardial Infarction Concerning Promptness in Taking Medical Aid. Herz Kreislauf, 19 (6), 307-314

Ponzillo, J.J. (1990), Focus on Ticlopidine - A Platelet-Aggregation Inhibitor for Stroke Prophylaxis. Hospital Formulary, 25 (5), 517-&

Gurwith, M. (1982), Infection and Stroke Following Mitral-Valve Replacement. Hospital Practice, 17 (1), U98-V98

Young, E.J., Reims, R.G. and Jolivet, D.A. (1983), Fever and Deterioration in Impaired Stroke Patient. Hospital Practice, 18 (10), 59-&

Stroh, J.A., Bagaria, S.K., Gabor, G.E. and Glassman, E. (1988), Tia in An Anticoagulated Patient A Dozen Years After A Stroke. Hospital Practice, 23 (7), 113-&

Lough, S., Wing, A.M., Fraser, C. and Jenner, J.R. (1984), Measurement of Recovery of Function in the Hemiparetic Upper Limb Following Stroke - A Preliminary-Report. Human Movement Science, 3 (3), 247-256

Sadoshima, S., Busija, D., Brody, M. and Heistad, D. (1981), Sympathetic-Nerves Protect Against Stroke in Stroke-Prone Hypertensive Rats - A Preliminary-Report. Hypertension, 3 (3), 124-127

Dietz, R., Schomig, A., Rascher, W., Strasser, R., Luth, J.B., Ganten, U. and Kubler, W. (1982), Contribution of the Sympathetic Nervous-System to the Hypertensive Effect of A High Sodium Diet in Stroke-Prone Spontaneously Hypertensive Rats. Hypertension, 4 (6), 773-781

Sadoshima, S. and Heistad, D. (1982), Sympathetic-Nerves Protect the Blood-Brain-Barrier in Stroke- Prone Spontaneously Hypertensive Rats. Hypertension, 4 (6), 904-907

Takeshita, A., Imaizumi, T., Ashihara, T. and Nakamura, M. (1982), Adrenergic-Mechanisms do Not Contribute to Salt-Induced Vasoconstriction in Stroke-Prone Spontaneously Hypertensive Rat. Hypertension, 4 (2), 288-293

Yamori, Y., Horie, R., Tanase, H., Fujiwara, K., Nara, Y. and Lovenberg, W. (1984), Possible Role of Nutritional Factors in the Incidence of Cerebral-Lesions in Stroke-Prone Spontaneously Hypertensive Rats. Hypertension, 6 (1), 49-53

Werber, A.H., Baumbach, G.L., Wagner, D.V., Mark, A.L. and Heistad, D.D. (1985), Factors That Influence Stroke in Dahl Salt-Sensitive Rats. Hypertension, 7 (1), 59-64

Bruner, C.A., Myers, J.H., Sing, C.F., Jokelainen, P.T. and Webb, R.C. (1986), Genetic Association of Hypertension and Vascular Changes in Stroke-Prone Spontaneously Hypertensive Rats. Hypertension, 8 (10), 904-910

Mohr, J.P. (1986), Lacunar Stroke. Hypertension, 8 (4), 349-356

Soltis, E.E. and Bohr, D.F. (1987), Vascular Reactivity in the Spontaneously Hypertensive Stroke- Prone Rat - Effect of Antihypertensive Treatment. Hypertension, 9 (5), 492-497

Sugimoto, T., Tobian, L. and Ganguli, M.C. (1988), High Potassium Diets Protect Against Dysfunction of Endothelial-Cells in Stroke-Prone Spontaneously Hypertensive Rats. Hypertension, 11 (6), 579-585

Lee, R.M.K.W., Nagahama, M., Mckenzie, R. and Daniel, E.E. (1988), Peptide-Containing Nerves Around Blood-Vessels of Stroke-Prone Spontaneously Hypertensive Rats. Hypertension, 11 (2), 117-120

Bruner, C.A. and Webb, R.C. (1988), Adrenal-Dependent Change in Vascular Reactivity in Stroke-Prone Spontaneously Hypertensive Rats. Hypertension, 12 (4), 388-392

Tomita, T., Ikeda, M., Inoue, Y., Mitubori, T. and Umegaki, K. (1989), Defective Protein-Phosphorylation Associated with Hypofunctions in Stroke-Prone Spontaneously Hypertensive Rat Platelets. Hypertension, 14 (3), 304-315

Okamoto, H., Kawaguchi, H., Minami, M., Saito, H. and Yasuda, H. (1989), Lipid Alterations in Renal Membrane of Stroke-Prone Spontaneously Hypertensive Rats. Hypertension, 13 (5), 456-462

Schober, M., Howe, P.R.C., Sperk, G., Fischercolbrie, R. and Winkler, H. (1989), An Increased Pool of Secretory Hormones and Peptides in Adrenal-Medulla of Stroke-Prone Spontaneously Hypertensive Rats. Hypertension, 13 (5), 469-474

Stier, C.T., Benter, I.F., Ahmad, S., Zuo, H., Selig, N., Roethel, S., Levine, S. and Itskovitz, H.D. (1989), Enalapril Prevents Stroke and Kidney Dysfunction in Salt-Loaded Stroke-Prone Spontaneously Hypertensive Rats. Hypertension, 13 (2), 115-121

Furspan, P.B. and Bohr, D.F. (1990), Calcium Sensitivity of Ca-2+-Activated K+ Channels in Spontaneously Hypertensive Stroke-Prone Rats. Hypertension,