References

104 (2), 235-241

Henrich, J.B. and Horwitz, R.I. (1989), The Contributions of Individual Factors to Thromboembolic Stroke. Journal of General Internal Medicine, 4 (3), 195-201

Hubert, P. and Mouget, G. (1981), Return Stroke Velocity-Measurements in 2 Triggered Lightning Flashes. Journal of Geophysical Research-Oceans and Atmospheres, 86 (NC6), 5253-5261

Master, M.J., Uman, M.A., Lin, Y.T. and Standler, R.B. (1981), Calculations of Lightning Return Stroke Electric and Magnetic- Fields Above Ground. Journal of Geophysical Research-Oceans and Atmospheres, 86 (NC12), 2127-2132

Idone, V.P. and Orville, R.E. (1982), Lightning Return Stroke Velocities in the Thunderstorm Research International Program (Trip). Journal of Geophysical Research-Oceans and Atmospheres, 87 (NC7), 4903-4915

Thomson, E.M., Galib, M.A., Uman, M.A., Beasley, W.H. and Master, M.J. (1984), Some Features of Stroke Occurrence in Florida Lightning Flashes. Journal of Geophysical Research-Atmospheres, 89 (ND3), 4910-4916

Uman, M.A. (1985), Lightning Return Stroke Electric and Magnetic-Fields. Journal of Geophysical Research-Atmospheres, 90 (ND4), 6121-6130

Mach, D.M. and Rust, W.D. (1989), Photoelectric Return-Stroke Velocity and Peak Current Estimates in Natural and Triggered Lightning. Journal of Geophysical Research-Atmospheres, 94 (D11), 13237-13247

Lanzerotti, L.J., Thomson, D.J., Maclennan, C.G., Rinnert, K., Krider, E.P. and Uman, M.A. (1989), Power Spectra at Radio-Frequency of Lightning Return Stroke Waveforms. Journal of Geophysical Research-Atmospheres, 94 (D11), 13221-13227

Rachidi, F. and Nucci, C.A. (1990), On the Master, Uman, Lin, Standler and the Modified Transmission-Line Lightning Return Stroke Current Models. Journal of Geophysical Research-Atmospheres, 95 (D12), 20389-20394

Cooray, V. and Orville, R.E. (1990), The Effects of Variation of Current Amplitude, Current Risetime, and Return Stroke Velocity Along the Return Stroke Channel on the Electromagnetic-Fields Generated by Return Strokes. Journal of Geophysical Research-Atmospheres, 95 (D11), 18617-18630

Rakov, V.A., Uman, M.A., Jordan, D.M. and Priore, C.A. (1990), Ratio of Leader to Return Stroke Electric-Field Change for 1St and Subsequent Lightning Strokes. Journal of Geophysical Research-Atmospheres, 95 (D10), 16579-16587

Keywords: SEP

Diendorfer, G. and Uman, M.A. (1990), An Improved Return Stroke Model with Specified Channel-Base Current. Journal of Geophysical Research-Atmospheres, 95 (D9), 13621-13644

Rakov, V.A. and Uman, M.A. (1990), Some Properties of Negative Cloud-To-Ground Lightning Flashes Versus Stroke Order. Journal of Geophysical Research-Atmospheres, 95 (D5), 5447-5453

Shadden, B.B. (1983), Pre-Crisis Intervention - A Tool for Reducing the Impact of Stroke-Related Personal and Family Crisis. Journal of Gerontological Social Work, 6 (1), 61-74

Tuomilehto, J., Piha, T., Nissinen, A., Geboers, J. and Puska, P. (1987), Trends in Stroke Mortality and in Antihypertensive Treatment in Finland from 1972 to 1984 with Special Reference to North Karelia. Journal of Human Hypertension, 1 (3), 201-208

Cruickshank, J.M., Higgins, T.J.C., Pennert, K., Thorp, J.M., Zacharias, F.M. and Zacharias, F.J. (1987), The Efficacy and Tolerability of Antihypertensive Treatment Based on Atenolol in the Prevention of Stroke and the Regression of Left-Ventricular Hypertrophy. Journal of Human Hypertension, 1 (2), 87-93

Tresserras, R., Serramajem, L., Canela, J., Armario, P., Pardell, H., Rue, M. and Salleras, L. (1990), Ecological Association Between Hypertension and Stroke in Catalonia (Spain) - Development and Use of An Ecological Regression-Model. Journal of Human Hypertension, 4 (4), 300-302

Keywords: HYPERTENSION

Ciccone, C.D. and Lyons, C.M. (1987), Relationships of Upper Extremity Strength and Swimming Stroke Technique on Competitive Freestyle Swimming Performance. Journal of Human Movement Studies, 13 (3), 143-150

Yamori, Y., Nara, Y., Mano, M. and Horie, R. (1985), Sympathetic Factors in the Cardiovascular Complications of Hypertension - Evidence in Genetic Models for Hypertension, Stroke and Atherosclerosis. Journal of Hypertension, 3 S35-S38

Tobian, L. (1986), High-Potassium Diets Markedly Protect Against Stroke Deaths and Kidney-Disease in Hypertensive Rats, An Echo from Prehistoric Days. Journal of Hypertension, 4 S67-S76

Memezawa, H., Katayama, Y., Sugimoto, S., Inamura, K., Shimizu, J., Soeda, T., Suzuki, S. and Terashi, A. (1986), Arachidonic Acid-Induced Brain Ischemia - Different Severity Between Stroke-Resistant Spontaneously Hypertensive Rats and Wistar-Kyoto Rats. Journal of Hypertension, 4 S421-S424

Sunano, S., Tsuji, A., Shimada, T. and Shimamura, K. (1986), Comparison of the Effects of Sodium Vanadate and Ouabain on Vascular Smooth Muscles of Wistar-Kyoto Rats and Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 4 S261-S262

Johansson, B.B. and Fredriksson, K. (1986), Cerebral Blood-Flow and Cerebrovascular Alterations in Spontaneously Hypertensive and Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 4 S89-S91

Ikemoto, F., Takada, T., Itoh, S., Miyazaki, M. and Yamamoto, K. (1986), Prevention of Development of Hypertension and Stroke in Stroke- Prone Spontaneously Hypertensive Rats by Chronic Treatment with Converting-Enzyme-Inhibitor Sa-446. Journal of Hypertension, 4 S499-S500

Tobian, L., Lange, J.M., Johnson, M.A., Macneill, D.A., Wilke, T.J., Ulm, K.M. and Wold, L.J. (1986), High-K Diets Markedly Reduce Brain Hemorrhage and Infarcts, Death Rate and Mesenteric Arteriolar Hypertrophy in Stroke- Prone Spontaneously Hypertensive Rats. Journal of Hypertension , 4 S205-S207

Suzuki, A., Higashino, H., Kondoh, S., Yamanishi, Y., Yoshimura, Y. and Niwa, A. (1986), Spare Receptors in the Blood-Vessels from Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 4 S77-S79

Moursi, M.G., Ganten, D., Lang, R.E. and Unger, T. (1986), Antihypertensive Action and Inhibition of Tissue Converting Enzyme (Ce) by 3 Prodrug Ce Inhibitors, Enalapril, Ramipril and Perindopril in Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 4 S495-S498

Okada, T., Kuwahara, T., Satoh, T. and Nakamura, K. (1986), Prevention of Ca-2+ Hypersusceptibility of Erythrocyte Cytoskeleton in Stroke-Prone Spontaneously Hypertensive Rats by A Calmodulin Antagonist. Journal of Hypertension, 4 S271-S273

Ikemoto, F., Itoh, S., Tanaka, M., Morita, N., Okamoto, K. and Yamamoto, K. (1986), Detection and Possible Mechanism of Formation of Renal High- Molecular-Weight Renin in Aged Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 4 S359-S360

Macmahon, S.W., Cutler, J.A., Neaton, J.D., Furberg, C.D., Cohen, J.D., Kuller, L.H. and Stamler, J. (1986), Relationship of Blood-Pressure to Coronary and Stroke Morbidity and Mortality in Clinical-Trials and Epidemiologic-Studies. Journal of Hypertension, 4 S14-S17

Yamada, S., Nakamoto, M., Hayashi, M., Tomita, T. and Hayashi, E. (1986), Tubular and Glomerular Adrenoceptors in Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 4 S209-S211

Randall, O.S., Westerhof, N., Vandenbos, G.C. and Alexander, B. (1986), Reliability of Stroke Volume to Pulse Pressure Ratio for Estimating and Detecting Changes in Arterial Compliance. Journal of Hypertension, 4 S293-S296

Nagaoka, A. (1986), Pharmacological Prevention of Cerebral Stroke in Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 4 S523-S525

Ohta, Y., Chikugo, T., Higashizawa, T., Chichibu, S. and Okamoto, K. (1986), A Study of the Relationship Between Arterial and Intracranial Hypertension in the M-Strain of Stroke-Prone Spontaneously Hypertensive Rats and Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 4 S409-S411

Umegaki, K., Ikeda, M., Nakamura, K., Inoue, Y. and Tomita, T. (1986), Platelet Dysfunction in Stroke-Prone Spontaneously Hypertensive Rats - Defective Ca-2+ Functions in Protein-Phosphorylation and in Coupling of Protein-Phosphorylation to Physiological- Responses. Journal of Hypertension, 4 S279-S281

Osol, G. and Halpern, W. (1986), Effect of Antihypertensive Treatment on Myogenic Properties of Brain Arteries from the Stroke-Prone Rat. Journal of Hypertension, 4 S517-S518

Fuchi, I., Noda, K. and Matsubara, Y. (1986), The Pregnant Stroke-Prone Spontaneously Hypertensive Rat As A Model Animal for Pregnancy Hypertension. Journal of Hypertension, 4 S375-S377

Kazda, S., Garthoff, B., Hirth, C., Preis, W. and Stasch, J.P. (1986), Parathyroidectomy Mimics the Protective Effect of the Calcium- Antagonist Nimodipine in Salt-Loaded Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 4 S483-S485

Okamoto, K., Yamamoto, K., Morita, N., Ohta, Y., Chikugo, T., Higashizawa, T. and Suzuki, T. (1986), Establishment and Use of the M-Strain of Stroke-Prone Spontaneously Hypertensive Rat. Journal of Hypertension, 4 S21-S24

Horino, N., Saitoh, H., Nishio, T., Mori, C., Horie, R. and Yamori, Y. (1986), Characteristics of Immunological Defect of Specific Pathogen- Free Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 4 S441-S443

Ueda, H., Koga, T., Akiba, T., Saito, N., Iwai, T. and Nakamura, M. (1986), Effects of Nipradilol, Propranolol and Pindolol on Myocardial Lesions in Salt-Loaded Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 4 S501-S503

Yamori, Y., Nara, Y., Tsubouchi, T., Sogawa, Y., Ikeda, K. and Horie, R. (1986), Dietary Prevention of Stroke and Its Mechanisms in Stroke-Prone Spontaneously Hypertensive Rats - Preventive Effect of Dietary Fiber and Palmitoleic Acid. Journal of Hypertension, 4 S449-S452

Nordborg, C. (1986), The Effects of Combined Metoprolol and Felodipine Treatment on the Arterial Morphometry of Spontaneously Hypertensive and Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 4 S509-S511

Ogawa, H., Shiota, C., Nishikawa, T., Fukushima, S. and Sasagawa, S. (1986), Age-Related-Changes in Serum Concentrations of Apolipoprotein- A-I, Apolipoprotein-e and Apolipoprotein-A-Iv in Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 4 S429-S431

Kalimo, H., Fredriksson, K., Nordborg, C., Johansson, B.B. and Olsson, Y. (1986), The Spread of Vasogenic Brain Edema and Its Role in Cerebral Tissue Destruction in Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 4 S407-S408

Paschen, W., Mies, G., Yamori, Y., Bodsch, W. and Hossmann, K.A. (1986), Regional Pattern of Hemodynamic and Biochemical Disturbances in Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 4 S403-S405

Suzuki, T., Yamamoto, J. and Minoda, M. (1986), Morphological-Study on the Relationship Between the Muscular Architecture of the Ventricles and Cardiac-Hypertrophy in the M-Strain of Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 4 S115-S118

Tagami, M., Kubota, A., Kitamura, J., Nara, Y. and Yamori, Y. (1986), Electron-Microscopic Studies of Ruptured and Occluded Arteries in Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 4 S413-S415

Furspan, P.B., Jokelainen, P.T., Sing, C.F. and Bohr, D.F. (1987), Genetic-Relationship Between A Lymphocyte Membrane Abnormality and Blood-Pressure in Spontaneously Hypertensive Stroke-Prone and Wistar-Kyoto Rats. Journal of Hypertension, 5 (3), 293-297

Kwan, C.Y., Seo, Y., Ito, H., Murakami, M. and Watari, H. (1987), High-Resolution Na-23 Nuclear-Magnetic-Resonance Study of Stroke-Prone Spontaneously Hypertensive Rat Erythrocytes. Journal of Hypertension, 5 (3), 359-366

Furspan, P.B. and Bohr, D.F. (1988), Effects of Felodipine on Blood-Pressure and Lymphocyte Membrane-Characteristics in Spontaneously Hypertensive Stroke- Prone Rats. Journal of Hypertension, 6 S236-S238

Thompson, L.E., Rinaldi, G.J. and Bohr, D.F. (1988), Sodium-Calcium Exchange in Vascular Smooth-Muscle of Wistar- Kyoto and Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 6 S160-S162

Shimizu, S., Nara, Y., Yamada, K., Keiser, H.R. and Yamori, Y. (1988), Cellular Mechanisms of Hypertension and Atherosclerosis - Hypoxia-Induced Lipid-Accumulation in Cultured Vascular Smooth- Muscle Cells from the Stroke-Prone Spontaneously Hypertensive Rat. Journal of Hypertension, 6 S163-S165

Arai, H., Nakao, K., Saito, Y., Sugawara, A., Yamada, T., Itoh, H., Mukoyama, M., Hosoda, K., Shirakami, G., Morii, N., Shiono, S., Ohkubo, H., Nakanishi, S. and Imura, H. (1988), Expression of Atrial Natriuretic Factor Gene in Hearts from Neonates of Spontaneously Hypertensive Rats and Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 6 S276-S278

Howe, P.R.C., Rogers, P.F. and Smith, R.M. (1989), Effects of Chronic Alcohol-Consumption and Alcohol Withdrawal on Blood-Pressure in Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 7 (5), 387-393

Howe, P.R.C., Rogers, P.F. and Head, G.A. (1989), Limited Baroreflex Control of Heart-Rate in Young Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 7 (1), 69-75

Bruner, C.A. and Webb, R.C. (1989), Effect of Felodipine on Blood-Pressure and Vascular Reactivity in Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 7 (1), 31-35

Macmahon, S. (1990), Antihypertensive Drug-Treatment - the Potential, Expected and Observed Effects on Vascular-Disease. Journal of Hypertension, 8 S239-S244.

Abstract: Re-analysis of the results of several prospective observational studies indicates that modest long-term differences in usual diastolic blood pressure are associated with larger differences in the primary incidence of stroke and of coronary heart disease than was previously believed. A prolonged difference of 5-6 mmHg in usual diastolic blood pressure was associated with at least one-third fewer strokes and at least one-fifth fewer coronary heart disease events. The results of randomly allocated trials of antihypertensive drug therapy (mostly diuretics) indicate that a similar difference in diastolic blood pressure maintained for just a few years appears to produce much or all of the potential long-term benefits for stroke. The incidence of coronary heart disease was also significantly reduced by treatment (2P < 0.01). However, for coronary disease, the trial results, even in combination, are inadequate to allow a reliable determination of the size of the treatment effects and the proportion of the long-term potential benefit achieved by a few years of diuretic-based treatment

Keywords: ANTIHYPERTENSIVE DRUGS/CORONARY HEART DISEASE/HYPERTENSION/RANDOMIZED TRIALS/STROKE/TRIAL/TRIALS

Lundjohansen, P., Omvik, P., White, W., Digranes, O., Helland, B., Jordal, O. and Stray, T. (1990), Long-Term Hemodynamic-Effects of Amlodipine at Rest and During Exercise in Essential-Hypertension. Journal of Hypertension, 8 (12), 1129-1136.

Abstract: The long-term haemodynamic responses to amlodipine, a new long- acting calcium antagonist, were studied both at rest and during exercise in 18 patients (mean age 43 years) with essential hypertension. Blood pressure was measured intra-arterially, cardiac output by dye dilution and heart rate by electrocardiogram. After 11 months of treatment with 5-10 mg amlodipine once daily (mean dose 9 mg/day), mean arterial pressure was reduced by 14% sitting at rest. The reduction in blood pressure was associated with a marked reduction in the total peripheral resistance index (TPRI) of 19% (P < 0.001). Similar responses were seen supine at rest and during 50 W, 100 W and 150 W bicycle exercise. No significant changes were seen in heart rate. There was a slight increase in stroke index, and cardiac index was preserved at rest and during exercise with a slight trend towards an increase. In 10 of the patients, blood pressure was monitored by a portable blood pressure recorder (Accutracker II, Suntech Medical Instruments, Raleigh, North Carolina, USA). Blood pressure was well controlled throughout the full 24 h period after one daily dose. In conclusion, amlodipine exerts a clear antihypertensive effect, both at rest and during exercise, through reduction in the TPRI and without a fall in cardiac pump function. No changes in heart rate were seen and there was no tendency for a reduction in the stroke index during 8 min of exercise at 150 W; on the contrary there was a trend towards an increase. The incidence of side-effects was low (ankle oedema in two patients)

Keywords: AMLODIPINE/ANTAGONISTS/BLOCKERS/BLOOD-PRESSURE/CALCIUM ANTAGONIST/ESSENTIAL HYPERTENSION/EXERCISE/HEMODYNAMICS/HYPERTENSION/THERAPY

Kobayashi, A., Nara, Y., Nishio, T., Mori, C. and Yamori, Y. (1990), Increased Na+-H+ Exchange Activity in Cultured Vascular Smooth- Muscle Cells from Stroke-Prone Spontaneously Hypertensive Rats. Journal of Hypertension, 8 (2), 153-157

Keywords: HYPERTENSION

Kaplan, N.M. (1990), Changing Hypertension Treatment to Reduce the Overall Cardiovascular Risk. Journal of Hypertension, 8 S175-S179.

Abstract: The increasing tendency to treat hypertension has markedly reduced stroke mortality but has not significantly reduced the mortality from coronary heart disease. Many explanations for this failure have been proposed and the true reason may never be known. Nevertheless, a number of steps can be taken to improve the degrees of therapeutic protection against coronary heart disease including (1) a complete assessment of all alterable risk factors; (2) effective use of multiple non-drug therapies; (3) use of antihypertensive drugs that reduce other cardiovascular risk factors; and (4) a gradual reduction of blood pressure to levels that will preserve perfusion to vital organs

Keywords: CORONARY HEART DISEASE/DIASTOLIC BLOOD-PRESSURE/HYPERTENSION/MYOCARDIAL-INFARCTION/NONDRUG THERAPY/RISK ASSESSMENT/STROKE MORTALITY/THERAPEUTIC PROTECTION

Lindblad, U., Rastam, L., Ryden, L., Ranstam, J., Berglund, G. and Isacsson, S.O. (1990), Reduced Stroke Incidence with Structured Hypertension Care - the Skaraborg Hypertension Project. Journal of Hypertension, 8 (12), 1147-1153.

Abstract: The Skaraborg Hypertension Project is a 5-year trial testing the impact of structured hypertension care implemented in half of the country, the other half serving as control. Population investigations revealed improved blood pressure control in all treated hypertensives in the study area, blood pressure reduction averaging 2-5 mmHg. The primary trial hypothesis of a possible impact on stroke incidence in the community was tested in this study. Cases of fatal and non-fatal stroke were identified from local and national mortality and morbidity registers, and diagnoses were validated against medical records. Of an initial 1169 reported cases, 1097 proved to be true stroke after validation. A statistically significant difference in stroke incidence trends (fatal and non-fatal) between the study and control populations was identified (P = 0.0251). Improved hypertension control in the study area seems the most plausible explanation for the relative decline in stroke incidence in the study area

Keywords: BLOOD-PRESSURE/COMMUNITY/HYPERTENSION/MORTALITY/NORTH-KARELIA/PRIMARY CARE/PROGRAM/STROKE/STRUCTURED CARE/TREATMENT GUIDELINES

Kannel, W.B. (1990), Implications of the Primary Prevention Trials Against Coronary Heart-Disease. Journal of Hypertension, 8 S245-S250.

Abstract: The bulk of the mortality (60%) in hypertension occurs in those with mild to moderate elevations of blood pressure, and the chief hazard is coronary disease. Although progression in the severity of hypertension has been slowed with drug therapy, the benefits for coronary outcome and all-cause mortality have been equivocal. Only a 10% reduction in coronary heart disease morbidity and mortality has been shown, an improvement that is not only small, but is statistically insignificant. Only vascular events such as renal failure, stroke, aortic dissection and cardiac failure have been reduced by antihypertensive therapy. Recent trials comparing beta-blockers with other antihypertensive drugs have failed to show the expected promise based on their effectiveness following a myocardial infarction. However, two large trials suggest that they may be effective against coronary heart disease in male non-smokers. A number of possible reasons for this therapeutic failure to reduce coronary heart disease have been postulated. The trials may have been to short to significantly affect the atherosclerotic progression. Also, sample sizes were too small to detect a sizeable reduction in coronary heart disease events. Furthermore, no attention was paid to improvement in the coronary heart disease risk profile, since drugs currently in use are known to have adverse effects on blood lipids, glucose tolerance and uric acid. It is even possible that a predisposition to sudden death is associated with antihypertensive therapy. The trials suggest that in attempts to prevent coronary heart disease, control of smoking and of serum lipids are particularly important in hypertensive persons and may be more effective than controlling the blood pressure alone. The trials suggest that we need greater emphasis on other antihypertensive agents. Also, non-pharmacologic methods for managing mild hypertension, such as salt restriction, control of obesity, increased exercise and a reduction in alcohol intake appear worthwhile. Since a large number of mild hypertensives must be treated to benefit a few, drug treatment should be targeted at those with a poor cardiovascular risk profile, where the bulk of events are concentrated. The treatment must be harmless and free of side effects that adversely affect the quality of life. Finally, trials in mild hypertension indicate that treatment must be multifactorial because blood pressure reductions alone are not enough

Keywords: ALCOHOL REDUCTION/BLOOD-PRESSURE/CORONARY HEART DISEASE/HYPERTENSION/INCREASED EXERCISE/MILD HYPERTENSION/MORBIDITY/MORTALITY/OBESITY/PRIMARY PREVENTION TRIALS/RISK PROFILE/SALT RESTRICTION/SERUM LIPIDS/SMOKING

Lindpaintner, K., Takahashi, S. and Ganten, D. (1990), Structural Alterations of the Renin Gene in Stroke-Prone Spontaneously Hypertensive Rats - Examination of Genotype- Phenotype Correlations. Journal of Hypertension, 8 (8), 763-773

Keywords: HYPERTENSION

Kobayashi, M., Morishita, H., Sugiyama, N., Yokochi, K., Nakano, M., Wada, Y., Hotta, Y., Terauchi, A. and Nonaka, I. (1986), Mitochondrial Myopathy, Encephalopathy, Lactic-Acidosis and Stroke-Like Episodes Syndrome and Nadh-Coq Reductase Deficiency. Journal of Inherited Metabolic Disease, 9 (3), 301-304

Maertens, P., Richardson, R., Bastian, F., Williams, J.P. and Hommes, F. (1988), A New Type of Mitochondrial Encephalomyopathy with Stroke-Like Episodes Due to Cytochrome-Oxidase Deficiency. Journal of Inherited Metabolic Disease, 11 186-188

Sandset, P.M. and Andersson, T.R. (1989), Coagulation Inhibitor Levels in Pneumonia and Stroke - Changes Due to Consumption and Acute Phase Reaction. Journal of Internal Medicine,